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Stimulated M2 polarization. Moreover, human explant mammary gland cultures treated with rSFRP1 show a marked reduction in the human M2 marker, CD163. CD163 is a monocyte/macrophage-restricted scavenge receptor [47] and the mechanism by which rSFRP1 reduces CD163 expression may be due in part to repression of Wnt signaling because Bergenfelz et al. show that breast cancer CD163+ TAMs correlate with
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Ed vaccines) or viruses (inactivated vaccines) inactivated by chemical or physicalEd vaccines) or viruses (inactivated vaccines) inactivated by chemical or physical treatmentsToxoids vaccinesPurified exotoxins chemically inactivated into toxoids that retain the ability to induce toxin-neutralizing antibodiesSubunit/ polysaccharide vaccinesAntigenic components of pathogens: partly or fully pu
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Ed vaccines) or viruses (inactivated vaccines) inactivated by chemical or physicalEd vaccines) or viruses (inactivated vaccines) inactivated by chemical or physical treatmentsToxoids vaccinesPurified exotoxins chemically inactivated into toxoids that retain the ability to induce toxin-neutralizing antibodiesSubunit/ polysaccharide vaccinesAntigenic components of pathogens: partly or fully pu
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In Figure 1. Case reading and reporting was mostly done by cardiologists (70 ), or a team of cardiologists and radiologists (26 ). Image quality was found to be good or excellent in 91 , moderate in 8 , and inadequate in 1 of cases. Severe complications occurred in a minority of patients (0.07 ) and were all associated with stress testing. We reported NSVT (n = 1), VF (n = 1) during dobutamine in
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Defect model in rabbits. The major merit of this study was that, this implant has been tested in vivo so that with a welldesigned pilot and experimental study we were able to explain the mechanism of action of this implant on tendon healing. In addition, we followed the immune activity of the body in response to the implanted collagen prosthesis from the early stages to four months after injury by
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Defect model in rabbits. The major merit of this study was that, this implant has been tested in vivo so that with a welldesigned pilot and experimental study we were able to explain the mechanism of action of this implant on tendon healing. In addition, we followed the immune activity of the body in response to the implanted collagen prosthesis from the early stages to four months after injury by
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Ased on NETPHOS software. We also includeAuthor Manuscript Author Manuscript Author Manuscript Author ManuscriptMatrix Biol. Author manuscript; available in PMC 2017 May 01.Boskey and Villarreal-RamirezPageSPARC (osteonectin), a protein involved with mineralization (linked in some ways to osteoporosis) that has some IDP regions. The SIBLINGs all bind to HA, however characterizing adsorption of pro
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